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VIP 6mg
Vilon 20mg (Bioregulator)
Vialox (Pentapeptide-3V) 200mg (Topical)
Vesugen 20mg (Bioregulator)
Vesilute 20mg (Bioregulator)
Triptorelin (GnRH) 2mg
Subtotal: $3,122.00
Free Shipping on All Orders Over $500
CJC-1295 (no DAC) (Modified GRF) is a truncated peptide analogue of growth hormone releasing hormone (GHRH). First developed in the 1980s, research studies with modGRF have shown it to improve muscle repair and growth, accelerate wound healing, strengthen bones, increase fat burning, and improve metabolism. It may also have beneficial effects on blood sugar regulation and the immune system.
CJC-1295 (No DAC), Ipamorelin, GHRP-2 (Blend)
CJC-1295 (no DAC), Hexarelin 10mg (Blend)
CJC-1295 (No DAC), GHRP-6 10mg (Blend)
CJC-1295 (No DAC), GHRP-2 10mg (Blend)
Chonluten is a short peptide bioregulator of gene expression. Research has shown it to be most active in lung tissue with secondary levels of activity in the GI tract. It regulates genes related to inflammation and antioxidant activity as well as genes involved in proliferation responses to inflammation. Regulation of pulmonary mucosal function by Chonluten may have important applications in chronic inflammatory diseases of the lung such as asthma and COPD.
Cartalax is a short bioregulatory peptide with widespread systemic impact due to its effects on fibroblasts. It has been shown to benefit skin and other connective tissues as well as the kidneys. Research shows that Cartalax increases cell proliferation rates and beneficially alters the levels of various cytokines and signaling molecules associated with senescence. Overall, it increases cell health and proliferation while decreasing cell death. Cartalax is thought of as an anti-aging peptide, returning older cells to a more youthful state and prolonging overall tissue health.
BPC-157, TB-500, KPV, GHK-Cu 80mg (Klow Blend)
BPC-157, TB-500, GHK-Cu 60mg (Glow Blend)
- BPC-157: A stable gastric pentadecapeptide shown to accelerate angiogenesis, fibroblast migration, and epithelial repair via modulation of VEGFR2, FAK-paxillin pathways, and nitric oxide signaling. It enhances tendon, muscle, and intestinal healing in preclinical models.
- TB-500 (Thymosin Beta-4): A 43-amino acid actin-sequestering peptide that promotes tissue regeneration through cell migration, angiogenesis (via VEGF upregulation), and anti-inflammatory effects. It mobilizes progenitor cells and accelerates repair of myocardium, dermis, and connective tissue.
- GHK-Cu: A copper-binding tripeptide (glycyl-L-histidyl-L-lysine) that stimulates wound healing, collagen synthesis, and hair growth. It modulates gene expression linked to tissue remodeling and exerts antioxidant and anti-inflammatory effects through TGF-β and metalloproteinase regulation.
- Angiogenesis: TB-500 and BPC-157 both promote VEGF-mediated vascularization, while GHK-Cu enhances endothelial cell proliferation.
- Cellular migration and matrix remodeling: TB-500 improves actin polymerization and cellular motility; GHK-Cu and BPC-157 stimulate ECM production and fibroblast activity.
- Anti-inflammatory modulation: All three reduce oxidative stress and cytokine-driven inflammation, potentially improving healing in chronic or complex injuries.
BPC-157, TB-500, GHK-Cu 30mg (Glow Blend)
- BPC-157: A stable gastric pentadecapeptide shown to accelerate angiogenesis, fibroblast migration, and epithelial repair via modulation of VEGFR2, FAK-paxillin pathways, and nitric oxide signaling. It enhances tendon, muscle, and intestinal healing in preclinical models.
- TB-500 (Thymosin Beta-4): A 43-amino acid actin-sequestering peptide that promotes tissue regeneration through cell migration, angiogenesis (via VEGF upregulation), and anti-inflammatory effects. It mobilizes progenitor cells and accelerates repair of myocardium, dermis, and connective tissue.
- GHK-Cu: A copper-binding tripeptide (glycyl-L-histidyl-L-lysine) that stimulates wound healing, collagen synthesis, and hair growth. It modulates gene expression linked to tissue remodeling and exerts antioxidant and anti-inflammatory effects through TGF-β and metalloproteinase regulation.
- Angiogenesis: TB-500 and BPC-157 both promote VEGF-mediated vascularization, while GHK-Cu enhances endothelial cell proliferation.
- Cellular migration and matrix remodeling: TB-500 improves actin polymerization and cellular motility; GHK-Cu and BPC-157 stimulate ECM production and fibroblast activity.
- Anti-inflammatory modulation: All three reduce oxidative stress and cytokine-driven inflammation, potentially improving healing in chronic or complex injuries.
BPC-157, TB-500 20mg (Blend)
PC-157 and TB-500 (Thymosin Beta-4) are peptides commonly studied for their potential in promoting tissue repair and healing. Both have distinct yet complementary mechanisms that, when combined, may offer synergistic benefits. BPC-157, a stable gastric pentadecapeptide, is derived from a protective protein found in the stomach lining. It has been shown in preclinical studies to accelerate healing of muscles, tendons, and ligaments by promoting angiogenesis (the formation of new blood vessels) and regulating the production of growth factors. Similarly, TB-500, a fragment of the naturally occurring Thymosin Beta-4 protein, plays a key role in cell migration, tissue regeneration, and reducing inflammation.
The potential synergy between these peptides arises from their overlapping yet distinct pathways in tissue repair. While BPC-157 excels at improving blood flow and reducing inflammation, TB-500 focuses on facilitating cellular migration to the injury site and supporting the repair process at the cellular level. Together, they may accelerate healing by addressing multiple aspects of tissue recovery, particularly in sports injuries or chronic conditions where regeneration is compromised. Early animal studies suggest this combination may lead to faster recovery times and improved structural integrity of repaired tissues.
It is crucial to note that while promising, much of the research on BPC-157 and TB-500 remains preclinical.
BPC-157, TB-500 10mg (Blend)
BPC-157 and TB-500 (Thymosin Beta-4) are peptides commonly studied for their potential in promoting tissue repair and healing. Both have distinct yet complementary mechanisms that, when combined, may offer synergistic benefits. BPC-157, a stable gastric pentadecapeptide, is derived from a protective protein found in the stomach lining. It has been shown in preclinical studies to accelerate healing of muscles, tendons, and ligaments by promoting angiogenesis (the formation of new blood vessels) and regulating the production of growth factors. Similarly, TB-500, a fragment of the naturally occurring Thymosin Beta-4 protein, plays a key role in cell migration, tissue regeneration, and reducing inflammation.
The potential synergy between these peptides arises from their overlapping yet distinct pathways in tissue repair. While BPC-157 excels at improving blood flow and reducing inflammation, TB-500 focuses on facilitating cellular migration to the injury site and supporting the repair process at the cellular level. Together, they may accelerate healing by addressing multiple aspects of tissue recovery, particularly in sports injuries or chronic conditions where regeneration is compromised. Early animal studies suggest this combination may lead to faster recovery times and improved structural integrity of repaired tissues.
It is crucial to note that while promising, much of the research on BPC-157 and TB-500 remains preclinical.




















